β-Amyloid peptides enhance α-synuclein accumulation and neuronal deficits in a transgenic mouse model linking Alzheimer's disease and Parkinson's disease
Proceedings of the National Academy of Sciences of the United States of America (2001) - Comments
pubmed: 11572944  doi: 10.1073/pnas.211412398  issn: 0027-8424  issn: 1091-6490 

Eliezer Masliah, Edward Rockenstein, Isaac Veinbergs, Yutaka Sagara, Margaret Mallory, Makoto Hashimoto, Lennart Mucke