β-Amyloid peptides enhance α-synuclein accumulation and neuronal deficits in a transgenic mouse model linking Alzheimer's disease and Parkinson's disease

Proceedings of the National Academy of Sciences (2001) - Comments
pubmed: 11572944 doi: 10.1073/pnas.211412398 issn: 0027-8424 issn: 1091-6490

Eliezer Masliah, Edward Rockenstein, Isaac Veinbergs, Yutaka Sagara, Margaret Mallory, Makoto Hashimoto, Lennart Mucke